NR507 Full Class Discussions
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NR507 Full Class Discussions
nr507 week 1 discussions
Week 1: Altered Immune System and Altered Inflammatory Response
– Discussion Part One
This week’s graded topics relate to the following Course Outcomes
(COs).
1 Analyze pathophysiologic mechanisms associated with selected
disease states. (PO 1)
2 Differentiate the epidemiology, etiology, developmental
considerations, pathogenesis, and clinical and laboratory manifestations of
specific disease processes. (PO 1)
3 Examine the way in which homeostatic, adaptive, and compensatory
physiological mechanisms can be supported and/or altered through specific
therapeutic interventions. (PO 1, 7)
6 Distinguish risk factors associated with selected disease
states. (PO 1)
5 Describe outcomes of disruptive or alterations in specific
physiologic processes. (PO 1)
6 Distinguish risk factors associated with selected disease
states. (PO 1)
7 Explore age-specific and developmental alterations in
physiologic
and disease states. (PO 1, 4)
Discussion
Discussion Part One (graded)
John is a 19-year-old college football player who presents with
sneezing, itchy eyes, and nasal congestion that worsens at night. He states
that he has a history of asthma, eczema and allergies
to pollen. There is also one other person on the football team
that has similar symptoms. His vitals are BP 110/70, P 84, R 18, T 100 F. Write
a differential of at least five (5) possible items from the most likely to less
likely. For each disease include information about the epidemiology,
pathophysiology and briefly argue why this disease fits the presentation and
why it might not fit the presentation
Week 1: Altered Immune System and Altered Inflammatory Response –
Discussion
Part Two
A patient has been admitted into the emergency room that was in
the passenger side of a car that collided with another car head on. The patient
is pale, barely conscious and has a weak and thready pulse. An IV is started.
The vitals are BP 80/50, P 140, T 96.0 and R 26. As the team fights to keep the
patient alive they have to remove the spleen. Blood is given but it has been
mistyped. A transfusion reaction occurs. Describe the type of hypersensitivity
reaction that has occurred and discuss the molecular pathophysiology of the
specific type of hypersensitive reaction you have chosen.
In the event that this patient survived the car accident and the
transfusion reaction which organs are most likely to be damaged and why?
Week 1: Altered Immune System and Altered Inflammatory
Response – Discussion Part Three
This week’s graded topics relate to the following Course Outcomes
(COs).
1 Analyze pathophysiologic mechanisms associated with selected
disease states. (PO 1)
2 Differentiate the epidemiology, etiology, developmental
considerations, pathogenesis, and clinical and laboratory manifestations of
specific disease processes. (PO 1)
3 Examine the way in which homeostatic, adaptive, and compensatory
physiological mechanisms can be supported and/or altered through specific
therapeutic interventions. (PO 1,
4 Distinguish risk factors associated with selected disease
states.(PO 1)
5 Describe outcomes of disruptive or alterations in specific
physiologic processes. (PO 1)
6 Distinguish risk factors associated with selected disease
states. (PO 1)
7 Explore age-specific and developmental alterations in
physiologic and disease states. (PO 1, 4)
Discussion
A 44-year-old patient presents with lump in the chest of
approximately 2 cm in diameter. There is a slight dimple over the location of
the lump and when the lump is manipulated it seems to be attached to the
surrounding tissue. A lumpectomy is performed and the mass is sent to
pathology. The pathology report comes back and the mass is confirmed to be an
estrogen receptor negative, a progesterone receptor negative and a her2/neu
receptor positive breast cancer.
What are some of the risk factors for breast cancer?
What tumor suppressor genes are associated with breast cancer?
What tumor oncogenes are associated with breast cancer?
Compare and contrast tumor suppressor genes from oncogenes?
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